Goldberger's Clinical Electrocardiography A Simplified Approach 10Th Edition pdf
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ECG SKILL DEVELOPMENT AND INCREASING DEMANDS FOR LITERACY
Throughout, we seek to stress the clinical applications and implications of ECG interpretation. Each time we mention an abnormal pattern, a clinical correlate is introduced. Although the book is not intended as a manual of therapeutics, we briefly discuss general principles of treatment and clinical management where relevant. Whenever possible, we have tried to put ourselves in the position of the clinician who has to look at ECGs without immediate specialist backup and make critical decisions—sometimes at 3 a.m.!
In this spirit, we have tried to approach ECGs in terms of a rational, simple differential diagnosis based on pathophysiology, rather than through the tedium of rote memorization. It is reassuring to discover that the number of possible arrhythmias that can produce a resting heart rate of more than 150 beats or more per minute is limited to just a handful of choices. Only three basic ECG patterns are observed during most cardiac arrests. Similarly, only a limited number of conditions cause low-voltage patterns, abnormally wide QRS complexes, ST segment elevations, and so forth.
ADDRESSING “THREE AND A HALF” KEY CLINICAL QUESTIONS
In approaching any ECG, readers should get in the habit of posing “three and a half ” essential queries: What does the ECG show and what else could it be? What are the possible causes of the waveform pattern or patterns? What, if anything, should be done about the finding(s)?
Most basic and intermediate-level ECG books focus on the first question (“What is it?”), emphasizing pattern recognition. However, waveform analysis is only a first step, for example, in the clinical diagnosis of atrial fibrillation. The following issues must always be addressed as part of answering the initial diagnostic question: What is the differential diagnosis? (“What else could it be?”) Are you sure that the ECG actually shows atrial fibrillation and not another “look-alike pattern,” such as multifocal atrial tachycardia (MAT), sinus rhythm with atrial premature beats, atrial flutter with variable block, or even an artifact resulting, for example, from parkinsonian tremor or a noisy baseline?
“What could have caused the arrhythmia?” is the question framing the next set of considerations. Is the atrial fibrillation associated with valvular or nonvalvular disease? If nonvalvular, is it related to hypertension, cardiomyopathy, coronary disease, advanced age, hyperthyroidism, or other factors, singly or in combination? On a deeper level are issues concerning the most basic electrophysiologic mechanisms. With atrial fibrillation, these mechanisms are still being worked out and involve a complex interplay of factors including abnormal pulmonary vein automaticity, micro-reentrant loops (wavelets) in the atria, inflammation and fibrosis (“atriopathy”), and autonomic perturbations
Finally, deciding on treatment and follow-up (“What are the therapeutic options and what is the best course to do choose in this case?”) depends in an essential way on answers to the questions posed above, with the ultimate goal of delivering the highest level of scientifically informed, compassionate care.
interactions where recognition of normal and abnormal patterns is only the starting point in patient care
The tenth edition contains updated discussions of multiple topics, including intraventricular and atrioventricular (AV) conduction disturbances, electronic pacemakers and implantable cardioverter–defibrillators (ICDs), sudden cardiac arrest, myocardial ischemia and infarction, takotsubo cardiomyopathy, atrial fibrillation and flutter, drug toxicities, amyloid cardiomyopathy, and COVID-19 infection, we highlight differential diagnoses, along with “pearls and pitfalls” in ECG interpretation. Familiarity with the limitations as well as the uses of the ECG is essential for novices and give special attention more seasoned clinicians. Reducing medical errors related to ECGs and maximizing the information content of these recordings, therefore, continue to be major themes
We also continue to give special emphasis to common points of confusion. Medical terminology (jargon) is rife with ambiguities that cause confusion and sometimes promote miscommunication. Students of electrocardiography face a barrage of challenges. Why do we call the “P-QRS interval” the “PR interval”? What is the difference between ischemia and injury? What is meant by the term “paroxysmal supraventricular tachycardia (PSVT)” and how does it differ (if it actually does) from “supraventricular tachycardia”? Is “complete AV heart block” synonymous with “AV dissociation”? I am delighted that the two coauthors on the previous two editions, Zachary D. Goldberger, MD, and Alexei Shvilkin, MD, PhD, continue in this role on the new tenth edition. We again thank our trainees and colleagues for their probing and challenging questions. Finally, we wish to express special gratitude to our families for their inspiration and encouragement. This edition again honors the memory of two remarkable individuals: the late Emanuel Goldberger, MD, a pioneer in the development of electrocardiography and the inventor of the aVR, aVL, and aVF leads, who was coauthor of the first five editions of this textbook (with ALG), and the late Blanche Goldberger, an exceptionally gifted artist and woman of valor.
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