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ZITELLI AND DAVIS,ATLAS OF Pediatric Physical Diagnosis,8th edition,2023

 

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PREFACE

In pediatrics, visual recognition of a variety of disorders is often the key factor in diagnosis. Experienced clinicians who have seen a wide spectrum of disorders carry a wealth of information for diagnosis and teaching. Despite the increased reliance on technology to make a diagnosis, history and physical examination remain the foundations of clinical assessment of the patient. The Atlas of Pediatric Physical Diagnosis was created to enhance the clinical experience of students, residents, nurses, and all practitioners who care for children. We have been very pleased with the reception of the first seven editions of the Atlas but frequently hear readers requesting new and updated information. Every chapter in the eighth edition has been reviewed, revised, and updated with the latest information. This edition also marks the first for our new co-editor, Dr. Jessica Garrison. Our new and returning chapter authors have provided greater depth and dimension while some sections have been extensively rewritten. New photographs and diagnostic images have been included in many chapters, and, where possible, we have cross-referenced common topics for readers to explore. Topical additions, such as COVID-19 infections in the Infectious Diseases chapter, represent our commitment to keeping the Atlas relevant to our changing clinical practice. The Atlas is by no means encyclopedic but rather presents an overview of clinical disorders that lend themselves to visual diagnosis. We have attempted to select disorders that are common or important and, when relevant, to describe the spectrum of clinical findings. The accompanying text deliberately emphasizes pertinent historical factors, examination techniques, visual findings, and diagnostic methods rather than therapy. We firmly believe that a careful history and physical examination are the best tools the clinician has for diagnosis and treatment. It is our hope that the eighth edition of the Atlas of Pediatric Physical Diagnosis continues to serve as a useful and practical reference for anyone who cares for children.

:CONTENTS

13 INFECTIOUS DISEASES 
13.1 Kernig and Brudzinski Signs

16 NEUROLOGY
16.1 Athetosis 
16.2 Breath Holding Spell 
16.3 Dystonia 
16.4 Hemifacial Spasms 
16.5 Childhood Onset Shuddering Attacks
 16.6 Rett Syndrome 
16.7 Sydenham Chorea 
16.8 Atonic Seizures 
16.9 Extensor Infantile Spasms 
16.10 Gelastic Seizures 
16.11 Generalized Tonic-Clonic Seizures 
16.12 Juvenile Myoclonic Seizures 
16.13 Non-epileptiform Seizure
 16.14 Absence Seizure 16.15 Complex Partial Seizure With Secondary Generalization 
16.16 Flexor Infantile Spasms 

17 PULMONARY DISORDERS
17.1 Anterior Tracheal Compression 
17.2 Bronchial Stenosis 
17.3 Layngomalacia 
17.4 Obstructive Apnea 
17.5 Vocal Cord Dysfunction Paradoxical Movement

19 PEDIATRIC AND ADOLESCENT GYNECOLOGY
19.1 Urination Through Prolapsed Urethra

20 OPHTHALMOLOGY 
20.1 Marcus Gunn Jaw Wink Phenomenon

24 OTOLARYNGOLOGY
24.1 Infected Preauricular Cyst 
24.2 Choanal Atresia 
24.3 Subglottic Stenosis 
24.4 Submucous Cleft Palate

GENETIC DISORDERS 1 AND DYSMORPHIC CONDITIONS
Suneeta Madan-Khetarpal | Georgianne Arnold | Damara Ortiz

The field of pediatric genetics and dysmorphology is complex, interesting, and rapidly evolving. Our knowledge base is gleaned from the careful observations of master clinicians and scientists who recognized clinical characteristics and patterns of malformation in individuals with genetic, teratogenic, developmental, and metabolic problems. They have provided us with a framework for the investigation of patients from clinical and laboratory perspectives. In addition to classic cytogenetics, molecular cytogenetics methods have been increasingly incorporated in clinical settings and have greatly assisted evaluation, enabling far greater understanding of the molecular and physiologic basis of these disorders, and have greatly increased the rate of diagnosis of children with genetic and metabolic disorders. However, even with the availability of an ever-widening array of confirmatory tests, clinical evaluation of patients remains an essential component of the complete assessment of children and adults with genetic diseases and dysmorphic conditions. This stems from the fact that careful evaluation can substantially reduce the number of differential diagnostic possibilities and, thereby, the number of diagnostic tests and the total expense. Visual identification of dysmorphic features, baseline anthropometrics combined with serial measurements with recognition of patterns of malformation and behavioral phenotypes, remains an integral part of the diagnostic algorithm. As in pediatrics in general, genetic disorders should be investigated on the basis of a careful history, with a family pedigree and a thorough physical examination including evaluation for the presence of major and minor anomalies, and thoughtful laboratory testing. This chapter is designed to present clinicians who care for children with background on the general principles of genetics and dysmorphology, as well as updated information about important advances in our field. Although not exhaustive, it provides a framework for the broad categories of genetic diseases and discusses an approach to the evaluation of the dysmorphic child. Definitions and examples of the types of disorders resulting in genetic and/or congenital anomalies in children are described, including malformations, deformations, disruptions, associations, and sequences. We include examples of disorders inherited through classic mendelian inheritance patterns, including single-gene mutations, such as Marfan syndrome, or de Lange syndrome, metabolic disorders such as phenylketonuria (PKU) or Smith-Lemli-Opitz syndrome, as well as examples of nonmendelian disorders, such as teratogenic exposures in utero and disruptions or deformations of previously normal fetal structures. Etiologic mechanisms of diseases, such as imprinting abnormalities, expansions of trinucleotide repeats in nuclear deoxyribonucleic acid (DNA), mitochondrial DNA, whole exome and genome sequencing, as well as the fields of -omics (metabolomics, proteomics, etc.), are introduced. As testing options grow, there is acknowledgement of the expanding role of genotype to phenotype correlation and its importance in the genomic era. 

APPROACH TO THE EVALUATION OF A DYSMORPHIC CHILD

Approximately 2% to 3% of liveborn infants have an observable physical structural abnormality (major anomaly). This number rises to about 4% to 5% by the time the child is old enough to attend school. Structural differences can be determined to be either major or minor in character
Major structural anomalies have functional significance. Examples are polydactyly, colobomas of the iris (see Chapter 20), meningomyelocele, and cleft lip. Minor anomalies are usually of cosmetic importance only. Examples are epicanthal folds of the eyes, single transverse palmar creases, and supernumerary nipples. The incidence of isolated major anomalies in the general newborn population is approximately 1%, and the incidence of minor anomalies is approximately 14%. Both are more common in premature newborns. The probability of an infant having a major anomaly increases with the number of minor anomalies found. Thus all children with multiple minor anomalies warrant a careful clinical assessment in order to find potentially significant occult major anomalies. Once an anomaly is identified, assessing its significance begins with a determination of whether the anomaly in question is a single localized error in morphogenesis or one component of a multiple malformation syndrome. An understanding of the pathophysiologic mechanisms that produce structural abnormalities or differences provides an opportunity to define the types of structural abnormalities seen. This also assists the process of identifying the etiology and arriving at a specific diagnosis, which then can be useful in determining the prognosis and estimating the risk of recurrence of a similar problem in future pregnancies. Definitions of the classifications of structural anomalies aid in communication between clinicians and in the process of evaluation and are summarized below:    1. Malformation: A malformation is an abnormality of embryonic morphogenesis of tissue. It usually results from genetic, chromosomal, or teratogenic influences, but it can be of multifactorial etiology. Malformations are divided into two main categories: (1) those that constitute a single primary defect in development and (2) those that represent a single component of a multiple malformation syndrome. A multiple malformation syndrome can be defined as one having several observed structural defects in development involving multiple organ systems that share the same known or presumed etiology. Malformations often require surgical intervention. 2. Deformation: A deformation represents an alteration (often molding) of an intrinsically normal tissue caused by exposure to unusual extrinsic forces. A classic example is clubfoot, which may be the result of uterine constraint from crowding associated with a multiple gestation. A more severe example is the compressed.









 

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